Template:Current week ecosystem agenda: Difference between revisions

Revision as of 08:15, 31 August 2020

2020 Week 36

Aug-31 Mo

Sep-01 Tu

A cell-permeable succinate prodrug can alleviate N-methyl carbamate-induced Complex I dysfunction

from the O2k-Network »US PA Philadelphia Kilbaugh T«, »US PA Philadelphia Jang DH« and »SE Lund Elmer E«

Janowska JI, Piel S, Saliba N, Kim CD, Jang DH, Karlsson M, Kilbaugh TJ, Ehinger JK (2020) Mitochondrial respiratory chain Complex I dysfunction induced by N-methyl carbamate ex vivo can be alleviated with a cell-permeable succinate prodrug carbamate toxicity and treatment. Toxicol In Vitro 65:104794. »Bioblast link«

Sep-02 We

Sep-03 Th

Sep-04 Fr

Sep-05 Sa

Sep-06 So

Abstract submission deadline - »ESSR 2020 Innsbruck AT«

@@ Line 1: / Line 1: @@
+Week 36
-Week 35
+* '''Aug-31 Mo'''
-* '''Aug-24 Mo'''
-:{|
-| width="110" align="center" | [[File:MitoPedia.jpg|left|80px|MitoPedia|link=MitoPedia]]
-|'''Can NADH be used as an externally added substrate to obtain the [[NADH electron transfer-pathway state]] in [[Mitochondrial_respiration |mitochondrial respiration]]? '''<br>
-No, in most types of intact mitochondria NADH cannot pass the [[mitochondrial inner membrane]] (mtIM). Yeast mitochondria are an exception. <br>
-- »[[Nicotinamide_adenine_dinucleotide|Nicotinamide adenine dinucleotide]]« and »[[MitoPedia:_Substrates_and_metabolites|Substrates and metabolites]]«
-|}
 ----
-* '''Aug-25 Tu'''
+* '''Sep-01 Tu'''
 :{|
 | width="100" align="center" |[[Image:O2k-Publications.jpg|70px|link=http://wiki.oroboros.at/index.php/O2k-Publications:_Topics |O2k-Publications in the MiPMap]]
-|'''Low-intensity exercise increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults'''<br>
+|'''A cell-permeable succinate prodrug can alleviate N-methyl carbamate-induced Complex I dysfunction'''<br>
-:from the O2k-Network »[[LV Riga Makrecka-Kuka M]]«
+:from the O2k-Network »[[US PA Philadelphia Kilbaugh T]]«, »[[US PA Philadelphia Jang DH]]« and »[[SE Lund Elmer E]]«
-*Liepinsh E, Makarova E, Plakane L, Konrade I, Liepins K, Videja M, Sevostjanovs E, Grinberga S, Makrecka-Kuka M, Dambrova M (2020) Low-intensity exercise stimulates bioenergetics and increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults. Physiol Rep 8:e14489. — [[Liepinsh 2020 Physiol Rep|»Bioblast link«]]
+*Janowska JI, Piel S, Saliba N, Kim CD, Jang DH, Karlsson M, Kilbaugh TJ, Ehinger JK (2020) Mitochondrial respiratory chain Complex I dysfunction induced by N-methyl carbamate ex vivo can be alleviated with a cell-permeable succinate prodrug carbamate toxicity and treatment. Toxicol In Vitro 65:104794. [[Janowska 2020 Toxicol In Vitro|»Bioblast link«]]
 |}
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-* '''Aug-26 We'''
+* '''Sep-02 We'''
-:{|
-| width="110" align="center" | [[File:MitoPedia.jpg|left|80px|MitoPedia|link=MitoPedia]]
-| '''Non-mitochondrial respiration or [[residual oxygen consumption]] (''Rox'')? — Concept-driven constructive terminology frames our perception.'''
-After inhibition of electron-transfer (ET) pathways or in the absence of fuel substrates, both ''mitochondrial'' non-ET reactions and ''non-mitochondrial'' reactions contribute to ''residual oxygen consumption'' in mt-preparations and living cells.  <br>
-- »'''[https://www.bioenergetics-communications.org/index.php/BEC_2020.1_doi10.26124bec2020-0001.v1 BEC: Mitochondrial physiology]'''« — Table 1: ''Rox'' induced by non-ET pathway oxidation reactions
-|}
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-* '''Aug-27 Th'''
+* '''Sep-03 Th'''
+----
+* '''Sep-04 Fr'''
+----
+* '''Sep-05 Sa'''
 ----
-* '''Aug-28 Fr'''
+* '''Sep-06 So'''
+::::* Abstract submission deadline - »[[ESSR 2020 Innsbruck AT]]«