Template:Current week ecosystem agenda: Difference between revisions
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* '''Aug-25 Tu''' | * '''Aug-25 Tu''' | ||
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| width="100" align="center" |[[Image:O2k-Publications.jpg|70px|link=http://wiki.oroboros.at/index.php/O2k-Publications:_Topics |O2k-Publications in the MiPMap]] | |||
|'''Low-intensity exercise increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults'''<br> | |||
:from the O2k-Network Β»[[LV Riga Makrecka-Kuka M]]Β« | |||
*Liepinsh E, Makarova E, Plakane L, Konrade I, Liepins K, Videja M, Sevostjanovs E, Grinberga S, Makrecka-Kuka M, Dambrova M (2020) Low-intensity exercise stimulates bioenergetics and increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults. Physiol Rep 8:e14489. [[Liepinsh 2020 Physiol Rep|Β»Bioblast linkΒ«]] | |||
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* '''Aug-26 We''' Β | * '''Aug-26 We''' Β |
Revision as of 10:34, 24 August 2020
2020 Week 35
- Aug-24 Mo
Can NADH be directly used as substrate to obtain the NADH electron transfer-pathway state in high-resolution respirometry (HRR)?
No, you cannot use NADH as a NADH-linked substrate for mitochondrial respiration since the mitochondrial inner membrane, which should be intact for HRR experiments, is not permeable to NADH.
- Β»Nicotinamide adenine dinucleotideΒ« and Β»Substrates and metabolitesΒ«
- Aug-25 Tu
Low-intensity exercise increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults
- from the O2k-Network Β»LV Riga Makrecka-Kuka MΒ«
- Liepinsh E, Makarova E, Plakane L, Konrade I, Liepins K, Videja M, Sevostjanovs E, Grinberga S, Makrecka-Kuka M, Dambrova M (2020) Low-intensity exercise stimulates bioenergetics and increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults. Physiol Rep 8:e14489. Β»Bioblast linkΒ«
- Aug-26 We
Non-mitochondrial respiration or residual oxygen consumption (Rox)? β Concept-driven constructive terminology frames our perception. After inhibition of electron-transfer (ET) pathways or in the absence of fuel substrates, both mitochondrial non-ET reactions and non-mitochondrial reactions contribute to residual oxygen consumption in mt-preparations and living cells.
- Β»BEC: Mitochondrial physiologyΒ« β Table 1: Rox induced by non-ET pathway oxidation reactions
- Aug-27 Th
- Aug-28 Fr