Template:Coupling-control tables: Difference between revisions
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File:Blue-book-cover 2020.jpg |link= | File:Blue-book-cover 2020.jpg |link=Gnaiger_2020_BEC_MitoPathways | The Blue Book 2020 | ||
File:Coupling control - mitochondrial and cellular respiratory rates.png |OXPHOS-, ROUTINE-, ET-, and LEAK states; respiratory capacities (''P'', ''R'', ''E'', ''L'') corrected for residual oxygen consumption ''Rox''. | File:Coupling control - mitochondrial and cellular respiratory rates.png |OXPHOS-, ROUTINE-, ET-, and LEAK states; respiratory capacities (''P'', ''R'', ''E'', ''L'') corrected for residual oxygen consumption ''Rox''. | ||
File:EPL-net and excess.jpg |4-compartmental OXPHOS model. (''1'') ET capacity ''E'' of the noncoupled electron transfer system ETS. OXPHOS capacity ''P'' is partitioned into (''2'') the dissipative LEAK component ''L'', and (''3'') ADP-stimulated ''P-L'' net OXPHOS capacity. (''4'') If ''P-L'' is kinetically limited by a low capacity of the phosphorylation system to utilize the protonmotive force ''pmF'', then the apparent ''E-P'' excess capacity is available to drive coupled processes other than phosphorylation P» (ADP to ATP) without competing with P». | File:EPL-net and excess.jpg |4-compartmental OXPHOS model. (''1'') ET capacity ''E'' of the noncoupled electron transfer system ETS. OXPHOS capacity ''P'' is partitioned into (''2'') the dissipative LEAK component ''L'', and (''3'') ADP-stimulated ''P-L'' net OXPHOS capacity. (''4'') If ''P-L'' is kinetically limited by a low capacity of the phosphorylation system to utilize the protonmotive force ''pmF'', then the apparent ''E-P'' excess capacity is available to drive coupled processes other than phosphorylation P» (ADP to ATP) without competing with P». | ||
Latest revision as of 18:53, 30 December 2020
The Blue Book 2020
OXPHOS-, ROUTINE-, ET-, and LEAK states; respiratory capacities (P, R, E, L) corrected for residual oxygen consumption Rox.
4-compartmental OXPHOS model. (1) ET capacity E of the noncoupled electron transfer system ETS. OXPHOS capacity P is partitioned into (2) the dissipative LEAK component L, and (3) ADP-stimulated P-L net OXPHOS capacity. (4) If P-L is kinetically limited by a low capacity of the phosphorylation system to utilize the protonmotive force pmF, then the apparent E-P excess capacity is available to drive coupled processes other than phosphorylation P» (ADP to ATP) without competing with P».
(P-L)/P is the OXPHOS P-L control efficiency. The biochemical coupling efficiency is independent of kinetic control by the phosphorylation system when expressed as the E-L coupling efficiency, (E-L)/E. (E-P)/E is the kinetic E-P control efficiency.
