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A list of all pages that have property "Description" with value "[[File:ROX.jpg|100px|link=https://wiki.oroboros.at/images/3/30/ROX.jpg]] '''Residual oxygen consumption''' ''Rox'' — respiration in the ROX state — is due to oxidative side reactions remaining after inhibition of the [[electron transfer pathway]] (ET pathway) in [[mitochondrial preparation]]s or living cells. Different conditions designated as ROX states (different combinations of inhibitors of CI, CII, CIII and CIV) may result in consistent or significantly different levels of oxygen consumption. Hence the best quantitative estimate of ''Rox'' has to be carefully evaluated. Mitochondrial respiration is frequently corrected for ''Rox'' as the [[baseline state]]. Then, total [[ROUTINE]], [[LEAK respiration]], [[OXPHOS]] or [[Electron transfer pathway |ET]] (''R'', ''L'', ''P'' and ''E'') respiration are distinguished from the corresponding ''Rox''-corrected, mitochondrial (ET-pathway linked) fluxes: ''R''(mt), ''L''(mt), ''P''(mt) and ''E''(mt). Alternatively, ''R'', ''L'', ''P'' and ''E'' are defined as ''Rox''-corrected rates, in contrast to total rates ''R''´, ''L''´, ''P''´ and ''E''´. When expressing ''Rox'' as a fraction of ET capacity ([[flux control ratio]]), total flux ''E''´ (not corrected for ''Rox''), should be taken as the reference. ''Rox'' may be related to, but is of course different from [[ROS]] production. In previous editions, (including [[Gnaiger 2020 BEC MitoPathways]]), the [[REN]] state was not distinguished from the ROX state. However, in novel applications (Q-Module and NADH-Module), a distinction of these states is necessary. Care must be taken when assuming ''Ren'' as a substitute of ''Rox'' correction of mitochondrial respiration.". Since there have been only a few results, also nearby values are displayed.

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Residual oxygen consumption + ([[File:ROX.jpg|100px|link=https://wiki.oro … [[File:ROX.jpg|100px|link=https://wiki.oroboros.at/images/3/30/ROX.jpg]] '''Residual oxygen consumption''' ''Rox'' — respiration in the ROX state — is due to oxidative side reactions remaining after inhibition of the [[electron transfer pathway]] (ET pathway) in [[mitochondrial preparation]]s or living cells. Different conditions designated as ROX states (different combinations of inhibitors of CI, CII, CIII and CIV) may result in consistent or significantly different levels of oxygen consumption. Hence the best quantitative estimate of ''Rox'' has to be carefully evaluated. Mitochondrial respiration is frequently corrected for ''Rox'' as the [[baseline state]]. Then, total [[ROUTINE]], [[LEAK respiration]], [[OXPHOS]] or [[Electron transfer pathway |ET]] (''R'', ''L'', ''P'' and ''E'') respiration are distinguished from the corresponding ''Rox''-corrected, mitochondrial (ET-pathway linked) fluxes: ''R''(mt), ''L''(mt), ''P''(mt) and ''E''(mt). Alternatively, ''R'', ''L'', ''P'' and ''E'' are defined as ''Rox''-corrected rates, in contrast to total rates ''R''´, ''L''´, ''P''´ and ''E''´. When expressing ''Rox'' as a fraction of ET capacity ([[flux control ratio]]), total flux ''E''´ (not corrected for ''Rox''), should be taken as the reference. ''Rox'' may be related to, but is of course different from [[ROS]] production.</br></br>In previous editions, (including [[Gnaiger 2020 BEC MitoPathways]]), the [[REN]] state was not distinguished from the ROX state. However, in novel applications (Q-Module and NADH-Module), a distinction of these states is necessary. Care must be taken when assuming ''Ren'' as a substitute of ''Rox'' correction of mitochondrial respiration.' correction of mitochondrial respiration.)

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