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Difference between revisions of "Kuznetsov 1999 Transplant Proc"

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(Created page with "{{Publication |title=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant. Proc. 31: 992. |a...")
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{{Publication
{{Publication
|title=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant. Proc. 31: 992.
|title=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant Proc 31:992.
|authors=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E
|info=[http://www.ncbi.nlm.nih.gov/pubmed/10083441 PMID: 10083441]
|authors=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger Erich
|year=1999
|year=1999
|journal=Transplant. Proc.
|journal=Transplant Proc
|mipnetlab=AT_Innsbruck_GnaigerE
|abstract=STUDIES of mitochondrial bioenergetics yield a quantitative assessment of cell injury after organ storage.<sup>1-4</sup>Β  To reduce artifacts of mitochondrial isolation, we optimized high-resolution respirometry with permeabilized cells and muscle fibers for investigation of mitochondrial cold ischemia/reperfusion (CIR) injury.
|abstract=STUDIES of mitochondrial bioenergetics yield a quantitative assessment of cell injury after organ storage.1–4 To reduce artifacts of mitochondrial isolation, we optimized high-resolution respirometry with permeabilized cells and muscle fibers for investigation of mitochondrial cold ischemia/reperfusion (CIR) injury.
|mipnetlab=AT Innsbruck Gnaiger E
|info=[http://www.ncbi.nlm.nih.gov/pubmed/10083441 PMID: 10083441]
|discipline=Mitochondrial Physiology, Biomedicine
}}
}}
{{Labeling
{{Labeling
|organism=Rat
|tissues=Heart
|preparations=Permeabilized cells, Permeabilized tissue
|injuries=Ischemia-reperfusion
|topics=Substrate
|couplingstates=OXPHOS
|instruments=Oxygraph-2k
|instruments=Oxygraph-2k
|discipline=Mitochondrial Physiology, Biomedicine
|discipline=Mitochondrial Physiology, Biomedicine
|organism=Rat
|tissues=Cardiac Muscle
|preparations=Isolated Mitochondria
|injuries=Ischemia-Reperfusion; Preservation
|topics=Respiration; OXPHOS; ETS Capacity, Coupling; Membrane Potential, Substrate; Glucose; TCA Cycle
}}
}}

Latest revision as of 04:24, 23 November 2021

Publications in the MiPMap
Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant Proc 31:992.

Β» PMID: 10083441

Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger Erich (1999) Transplant Proc

Abstract: STUDIES of mitochondrial bioenergetics yield a quantitative assessment of cell injury after organ storage.1-4 To reduce artifacts of mitochondrial isolation, we optimized high-resolution respirometry with permeabilized cells and muscle fibers for investigation of mitochondrial cold ischemia/reperfusion (CIR) injury.


β€’ O2k-Network Lab: AT Innsbruck Gnaiger E


Labels:

Stress:Ischemia-reperfusion  Organism: Rat  Tissue;cell: Heart  Preparation: Permeabilized cells, Permeabilized tissue 

Regulation: Substrate  Coupling state: OXPHOS 

HRR: Oxygraph-2k