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This page provides a simple browsing interface for finding entities described by a property and a named value. Other available search interfaces include the page property search, and the ask query builder.

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A list of all pages that have property "MiPNet keyword" with value "Alzheimer's disease (AD)". Since there have been only a few results, also nearby values are displayed.

Showing below up to 42 results starting with #1.

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List of results

    • US KS Kansas City Wilkins H  + (Alzheimer’s Disease, brain bioenergetics)
    • FR Pessac Pasdois P  + (Atrial fibrillation, sudden death, energetic metabolism, mitochondrial permeability transition pore)
    • US FL Gainesville Wohlgemuth SE  + (Autophagy and mitochondrial function, skeletal and heart muscle)
    • NL Amsterdam Nollet E  + (Bio-energetics, heart failure, cardiac muscle, skeletal muscle, oxidative stress)
    • NL Amsterdam Wuest RC  + (Bio-energetics, skeletal muscle, cardiac muscle, metabolomics)
    • US LA New Orleans Katakam PV  + (Bioenergetics of neurons and cerebral microvasculature)
    • SE Gothenburg Hagberg H  + (Brain injury and regeneration - experimental studies and mechanisms)
    • CZ Prague Fisar Z  + (Brain mitochondrial bioenergetics, effect of psychotropic drugs on mitochondrial bioenergetics, mitochondrial dysfunctions in mood disorders and in Alzheimer’s disease)
    • IT Florence Morandi A  + (Cancer Cells, Bioenergetics, Metabolic reprogramming, mitochondria)
    • IT Rome Scatena R  + (Cancer cells and mitochondria)
    • UK Oxford Timm K  + (Cardio-oncology, namely in the side effect … Cardio-oncology, namely in the side effects of chemotherapy on the heart. Particularly interested in how different classes of chemotherapeutics affect cardiac metabolism and energetics, and how cardioprotective agents may interfere with, or potentiate, anti-cancer action of chemotherapeutics.Use the O2k for HRR of cardiac muscle fibres, isolated cardiac mitochondria, tumour homogenates and cardiomyocytes and cancer cells. Have collaborations in neuropharmacology and are happy to discuss potential collaborations across disciplinesotential collaborations across disciplines)
    • FR Montpellier Prouteau-Angebault C  + (Cardiolipin, cytochrome c oxidase, OPA 1)
    • US CO Fort Collins Chicco AJ  + (Cardiometabolic disease, environmental stresss, cardiolipin, comparative biochemistry)
    • NO Trondheim Hoydal MA  + (Cellular and molecular cardiology, ischemic heart disease, Exercise medicine)
    • CH Lausanne Place N  + (Cellular mechanisms underpinning skeletal muscle adaptations to exercise, Calcium handling, Neuromuscular plasticity)
    • US NC Durham Li PA  + (Cerebral ischemia, Nutrition, Mitochondrial biogenesis)
    • CA Sherbrooke Blondin DP  + (Cold-induced thermogenesis, heat exposure, exercise, type 2 diabetes, sepsis, cardiomyopathy)
    • BR Manaus Val AL  + (Comparative mitochondrial physiology)
    • US LA Baton Rouge Hand SC  + (Comparative physiology and biochemistry, animal bioenergetics, adaptations to anoxia and water stress, mitochondria physiology, Late embryogenesis Abundant (LEA) proteins)
    • US NC Greenville Neufer PD  + (Diabetes, Obesity, Insulin resistance, Exercise, Fat oxidation, Skeletal muscle)
    • US AL Birmingham Moellering DR  + (Diabetes, Obesity, Mitochondrial Physiology, Nutritional Bioenergetics, Redox Biology)
    • SE Uppsala Liss P  + (Diabetes, kidney)
    • US KS Kansas City Morris EM  + (Dr. Morris investigates how the susceptibi … Dr. Morris investigates how the susceptibility to diet-induced weight gain is mediated by systemic and peripheral energy metabolism through the modulation of the neural centers regulating energy intake and expenditure. His work centers on how the function of the primary energy producing cellular bodies, mitochondria, can impact tissue function and ultimately systemic health. Currently, the lab is investigating: 1) how liver mitochondrial function can, through neural pathways to the brain, influence high fat diet-induced weight, 2) how systemic energy expenditure serves as an independent regulator of weight gain and adiposity, and 3) whether transcriptional control of mitochondria in the ventromedial hypothalamus is necessary for susceptibility to weight gain.cessary for susceptibility to weight gain.)
    • FI Tampere Dufour E  + (Drosophila, Aging, Mitochondria, Complex I, NADH)
    • CA Rimouski Blier PU  + (Drosophila, Temperature adaptation)
    • US TX Denton Dzialowski EM  + (Endothermy, avian, reptile, cardiac, skeletal, liver, hypoxia)
    • US OK Stillwater Davis MS  + (Exercise Physiology, Respiratory Physiology, Hypoxia)
    • US CA Los Angeles Shirihai O  + (Exercise physiology, obesity)
    • US NC Durham Koves TR  + (Exercise, Heart Failure, Ketones, Lipid Oxidation, Metabolomics)
    • US OR Corvallis Robinson MM  + (Exercise, insulin resistance, obesity, humans, mouse model)
    • CA Antigonish Kane DA  + (Exercise, skeletal muscle, metabolism, diabetes, obesity)
    • AT Innsbruck Jansen-Duerr P  + (FAHD1, senescence, aging, ''Mus musculus'', ''C. elegans'')
    • US SD Brookings Baiao Menezes AC  + (Fetal programming, Efficiency of energy utilization in beef cattle)
    • BR Rio de Janeiro Oliveira MF  + (Heme metabolism, redox biochemistry, energy metabolism, mitochondrial function, Schistosoma mansoni)
    • CA London Staples JF  + (Hibernation, torpor, mitochondria)
    • US CO Colorado Springs Jacobs RA  + (Human bioenergetics in health and disease; Human Performance; Environmental effects on biological thermodynamics (emphasis on hypoxia/high altitude); and Aging)
    • US TX Galveston Sheffield-Moore M  + (Human muscle metabolism, cancer, aging)
    • CZ Prague Krajcova A  + (Human skeletal, cardiac muscle, mitochondrial disorders and mitochondrial pharmacology.)
    • HU Budapest Tretter L  + (Hydrogen peroxide, Synaptosomes, Guinea pigs, Mitochondrion, Reactive oxygen species, Oxidative stress)
    • NZ Auckland Hickey AJ  + (Hypoxia, Thermal limits on life, Heart disease, Ocean acidification, Spectroscopy, Reactive oxygen species, Oxidative stress)
    • UK Cambridge Murray AJ  + (Hypoxia, high altitude, heart failure, skeletal muscle, placenta)