Cannon B

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COST Action CA15203 (2016-2021): MitoEAGLE
Evolution-Age-Gender-Lifestyle-Environment: mitochondrial fitness mapping


Cannon B


MitoPedia topics: EAGLE 

COST: Member


COST WG3: WG3

COST Mentor: Mentor

MC Member - Management Committee MitoEAGLE
Name Cannon Barbara, Prof., Dr.
Institution Department of Metabolic Research
Barbara Cannon

The Wenner-Gren Institute

Stockholm University

Address Svante Arrhenius vΓ€g 20C

Room F350, 106 91

City Stockholm
State/Province
Country Sweden
Email [email protected]
Weblink
O2k-Network Lab SE Stockholm Nedergaard J


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Publications

 PublishedReference
Shabalina 2024 Biochim Biophys Acta Bioenerg2024Shabalina IG, JimΓ©nez B, Sousa-Filho CPB, Cannon B, Nedergaard J (2024) In isolated brown adipose tissue mitochondria UCP1 is not essential for nor involved in the uncoupling effects of the classical uncouplers FCCP and DNP. Biochim Biophys Acta Bioenerg [Epub ahead of print]. https://doi.org/10.1016/j.bbabio.2024.149516
Gao 2022 Biochim Biophys Acta Bioenerg2022Gao Y, Shabalina IG, Braz GRF, Cannon B, Yang G, Nedergaard J (2022) Establishing the potency of N-acyl amino acids versus conventional fatty acids as thermogenic uncouplers in cells and mitochondria from different tissues.
Nabben 2011 Biochim Biophys Acta2011Nabben M, Shabalina IG, Moonen-Kornips E, van Beurden D, Cannon B, Schrauwen P, Nedergaard J, Hoeks J (2011) Uncoupled respiration, ROS production, acute lipotoxicity and oxidative damage in isolated skeletal muscle mitochondria from UCP3-ablated mice. Biochim Biophys Acta 1807:1095-105.
Cannon 2008 Methods Mol Biol2008Cannon B, Nedergaard J (2008) Studies of thermogenesis and mitochondrial function in adipose tissues. Methods Mol Biol 456:109-21.

Abstracts

 PublishedReference
Gao 2019 MiPschool Coimbra2019
MiPsociety
Effects on mitochondrial respiration of N-acyl amino acids versus common fatty acids.
Cannon 2019 MiP20192019
Barbara Cannon
No Abstract
Shabalina 2017 Abstract MITOEAGLE Barcelona2017
COST Action MITOEAGLE
Control of UCP1 activity in brown fat mitochondria.
Shabalina 2017 MiP20172017
Irina Shabalina
In UCP1-ablated mice, prolonged cold exposure promotes mitochondrial dysfunction, apoptosis and inflammation in brown adipose tissue, and accumulation of visceral obesogenic fat.
Nedergaard 2017 MiP20172017
Jan Nedergaard
Creatine effect in mitochondria from different tissues.
Kalinovich 2015 Abstract MiP20152015UCP3-related changes in brown and brite/beige adipose tissue in cold-acclimated mice.
Shabalina 2015 Abstract MiP20152015The metabolically inert environmental pollutants perfluorinated fatty acids directly activate uncoupling protein 1 in brown-fat mitochondria.
Mahdaviani 2014 Abstract MiP20142014Hormone‐induced mitochondrial fission is utilized by brown adipocytes as an amplification pathway for energy expenditure.
Shabalina 2013 Abstract IOC792013Shabalina IG (2013) Enhanced Complex I but low Complex II substrate-derived ROS production rates in mice, prematurely aging due to enhanced mitochondrial DNA mutation rate. Mitochondr Physiol Network 18.07
Shabalina 2013 Abstract MiP20132013Shabalina IG, Petrovic N, Kalinovich AV, Cannon B, Nedergaard J (2013) Comparative study of brown and white adipose tissue mitochondria in mice upon cold acclimation. Mitochondr Physiol Network 18.08.


Participated at

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