Petit Patrice X: Difference between revisions
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Revision as of 14:35, 24 September 2013
Name | Petit Patrice X, Dr. |
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Institution |
Toxicologie, Pharmacologie et Signalisation Cellulaire INSERM S-747 Centre Universitaire des Saints-Pères |
Address | 45 Rue des Saints-Pères, 75270 |
City | Paris |
State/Province | |
Country | France |
Weblink | |
O2k-Network Lab |
Labels:
Publications
Published | Reference | |
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BEC 2020.1 doi10.26124bec2020-0001.v1 | 2020 | Gnaiger E et al β MitoEAGLE Task Group (2020) Mitochondrial physiology. Bioenerg Commun 2020.1. https://doi.org/10.26124/bec:2020-0001.v1 |
Abstracts
Published | Reference | |
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Petit 2019 MiP2019 | 2019 | No abstract, Co-autor of Rainey 2019 MiP2019. |
Rainey 2019 MiP2019 | 2019 | Mitochondria are not the primary target of curcumin-induced autophagy and apoptosis. |
Petit 2018 MiP2018 | 2018 | Decreasing cytosolic translation is beneficial to yeast and human Tafazzin-deficient cells with dirupted mitochondrial homeostasis (Barth syndrome cell model). |
Petit 2017 MiP2017 | 2017 | Barth syndrome cardiolipin alterations linked to tafazzin mutations lead to apoptosis and mitophygy alterations. |
Petit 2013 Abstract MiP2013 | 2013 | Petit PX (2013) The cardiolipin defect linked to a tafazzin mutation in Barth syndrome disturbs mitochondrial metabolism, alters apoptosis and autophagic fluxes but does not affect cell cycle progression. Mitochondr Physiol Network 18.08. |