Shabalina IG

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Evolution-Age-Gender-Lifestyle-Environment  » COST Action CA15203 Mitochondrial fitness mapping

Shabalina IG


MitoPedia topics: EAGLE 

COST: Member COST WG1: WG1

COST WG3: WG3


Name Shabalina Irina G., Dr.
Institution
Irina G Shabalina

The Wenner-Gren Institute

Stockholm University

Address The Arrhenius Laboratories F3, SE-10691
City Stockholm
State/Province
Country Sweden
Email irina.shabalina@wgi.su.se
Weblink
O2k-Network Lab SE Stockholm Nedergaard J


Labels: Field of research: Basic  Topics: uncoupling proteins, mitochondria, brown and white adipose tissue, mtDNA mutation, mitochondrial ROS production 


Publications

Published Reference
Nabben 2011 Biochim Biophys Acta 2011 Nabben M, Shabalina IG, Moonen-Kornips E, van Beurden D, Cannon B, Schrauwen P, Nedergaard J, Hoeks J (2011) Uncoupled respiration, ROS production, acute lipotoxicity and oxidative damage in isolated skeletal muscle mitochondria from UCP3-ablated mice. Biochim Biophys Acta 1807:1095-105.
Fernstroem 2007 J Appl Physiol (1985) 2007 Fernström M, Bakkman L, Tonkonogi M, Shabalina IG, Rozhdestvenskaya Z, Mattsson CM, Enqvist JK, Ekblom B, Sahlin K (2007) Reduced efficiency, but increased fat oxidation, in mitochondria from human skeletal muscle after 24-h ultraendurance exercise. J Appl Physiol 102:1844–9.


Abstracts

Published Reference
Shabalina 2015 Abstract MiP2015 2015 The metabolically inert environmental pollutants perfluorinated fatty acids directly activate uncoupling protein 1 in brown-fat mitochondria.
Kalinovich 2015 Abstract MiP2015 2015 UCP3-related changes in brown and brite/beige adipose tissue in cold-acclimated mice.
Shabalina 2013 Abstract MiP2013 2013 Shabalina IG, Petrovic N, Kalinovich AV, Cannon B, Nedergaard J (2013) Comparative study of brown and white adipose tissue mitochondria in mice upon cold acclimation. Mitochondr Physiol Network 18.08.
Shabalina 2013 Abstract IOC79 2013 Shabalina IG (2013) Enhanced Complex I but low Complex II substrate-derived ROS production rates in mice, prematurely aging due to enhanced mitochondrial DNA mutation rate. Mitochondr Physiol Network 18.07

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